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Scale ; compared with age-matched controls without RLS odds ratio [OR] 13.06; 95% confidence interval [CI] 2.32-73.26 ; . The diagnosis of RLS by trained physicians in this study is important, as there are many mimics of RLS that can create false-positive diagnoses, 1 of which could be depression itself. However, other potential confounders eg, other medical disorders or pain ; were not controlled for in the logistic regression. Similarly, a Japanese population-based study of 2023 elderly individuals who responded to questionnaires regarding depression, sleep, and RLS found that men between the ages of 65 and 75 years were 3.6 times 95% CI 1.2-10.6 ; more likely to have elevations in depression scores by the Geriatric Depression Scale ; if they had probable RLS.19 Simple and multiple logistic regression analysis was done. Women with RLS, and those over the age of 75 years, did not demonstrate an elevated risk of depression scores with RLS. In another population-based study which also controlled for confounding variables ; utilizing questionnaires returned by more than 2608 Swedish men, Ulfberg et al found that those with RLS more frequently reported "depressed mood" than did those without RLS OR 2.6, 95% CI 1.8-3.8 ; .20 Other symptoms of depression--including decreased concentration, irritability, fatigue, and decreased libido--were also statistically more common in those with RLS. In a group of 103 Turkish adults with RLS derived from a community sample of 3234, Hamilton Depression Scale scores were found to be significantly higher than in controls P .001 ; , even when the 3 sleep questions in this Scale were omitted, with a positive correlation between severity of RLS and depression symptoms r 0.201, P .04 ; .21 The interviews were face to face, but comparison was with 103 age- and sex-matched controls rather than the surveyed population as a whole. Depression scores were not found to vary with sex, cigarette smoking, income status, or existence of any comorbid disease. Although depression symptoms were not specifically addressed, 1 other population-based study examined the association of RLS diagnosed by a single question ; and self-described "poor mental health" in 1803 adults in Kentucky. Those reporting symptoms consistent with RLS were 3.1 times 95% CI 2.0-4.6 ; more likely to report poor mental health on all of the previous 30 days than were those who did not have such symptoms.22 Nine studies examined individuals diagnosed with RLS in a clinical setting, who were then compared to noncomplaining populations, to historical controls, or in 2 studies to other hemodialysis patients. Winkelmann et al administered DSM-IVbased depression and anxiety questionnaires M-CIDI DIA-X ; to 130 RLS patients and found higher 12-month rates of a major depressive episode, compared with the control population 17.7% vs 8.7%; OR 2.55; 95% CI 1.5-4.4 ; .23 Lifetime prevalence rates of major depression also exceeded controls 36.9% vs 15.2%; OR 3.30, 95% CI 2.1-5.0 ; , with the majority 77% ; reporting the onset of depression after the onset of RLS. Vandeputte and de Weerd reported elevated Beck Depression Inventory scores in 53% of 154 consecutive sleep clinic patients with RLS or PLMD.24 This was in spite of patients with "clinically overt depression" being excluded from the study. Comparing 33 adults with RLS to controls, Saletu et al25 found higher depression scores on the Zung Self-Rating Depression Scale in the RLS group mean scores 39.9 and 29.6 respectively, P .001 ; . Patients requiring psychoactive medication were excluded from the study. Of 55 RLS patients seen in a Swiss sleep clinic, 33% described "depression symptoms" at some point during their life, and several reported exacerbation of.
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Parenteral nutrition - intravenous infusion of some or all of a patient's nutritional requirements through a catheter fine tube ; placed in the vein. See also hyperalimentation ; . pathogen - harmful organism eg: bacterium, virus ; causing disease. pathologist - a doctor who is a specialist in the examination of tissue. See also histopathologist ; . perforation - an abnormal opening in the bowel wall which causes the contents of the bowel to leak into the normally sterile abdominal cavity. perianal - the area round the anal opening i.e. around the anus ; . peritoneum - the membrane lining the inside of the abdominal cavity. peritonitis - inflammation of the peritoneum often due to a perforation of the wall of the intestine. piles haemorrhoids ; - swollen veins in the area of the anus which bleed easily and can become painful. polyp - a growth protruding from mucosa. pouch ileo-anal ; - an internal pouch or reservoir made from the lower part of the intestine ileum ; which is attached to the anus, therefore maintaining continence and enabling the passage of stools in the normal manner. pouchitis - inflammation of an ileoanal pouch. prednisolone - is a drug of the corticosteroid group which is used to reduce inflammation in IBD. It can be taken orally as tablets, intravenously by injection, or through the rectum by an enema or suppository. proctitis inflammation situated about the rectum or anus. proctocolectomy total colectomy ; - the surgical removal of the colon and rectum. Proctosigmoiditis inflammation of the rectum and lower colon. prognosis - a prediction of what might happen in the future ie: the likely progress of the disease. prophylactic therapy - preventive treatment. proximal - further up the bowel towards the mouth. purinethol - see 6-mercaptopurine. pus a thick white, yellow or greenish fluid found in abscesses, on ulcers, and on inflamed or discharging surfaces.

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Broad-spectrum -lactamases conferring transferable resistance to newer -lactam agents in Enterobacteriaceae: hospital prevalence and susceptibility patterns. Rev. Infect. Dis. 10: 867878. 14. Katsanis, G. P., J. Spargo, M. J. Ferraro, L. Sutton, and G. A. Jacoby. 1994. Detection of Klebsiella pneumoniae and Escherichia coli strains producing extended-spectrum -lactamases. J. Clin. Microbiol. 32: 691696. 14a.Lofland, D., and L. A. Weymouth. Unpublished data. 15. Meyer, K. S., C. Urban, J. A. Eagan, B. J. Berger, and J. J. Rahal. 1993. Nosocomial outbreak of Klebsiella infection resistant to late-generation cephalosporins. Ann. Intern. Med. 119: 353358. 16. National Committee for Clinical Laboratory Standards. 1993. Performance standards for antimicrobial disk susceptibility tests, 5th ed. Approved standard M2-A5. National Committee for Clinical Laboratory Standards, Villanova, Pa. 17. National Committee for Clinical Laboratory Standards. 1993. Methods for dilution antimicrobial susceptibility tests for bacteria that grow aerobically, 3rd ed. Approved standard M7-A3. National Committee for Clinical Laboratory Standards, Villanova, Pa. 18. National Committee for Clinical Laboratory Standards. 1995. Performance standards for antimicrobial susceptibility testing, 6th informational supplement. M100-S5. National Committee for Clinical Laboratory Standards, Villanova, Pa. 19. Naumovski, L., J. P. Quinn, D. Miyashiro, M. Patel, K. Bush, S. B. Singer, D. Graves, T. Palzkill, and A. M. Arvin. 1992. Outbreak of ceftazidime resistance due to a novel extended-spectrum -lactamase in isolates from cancer patients. Antimicrob. Agents Chemother. 36: 19911996. 20. Philippon, A., S. Ben Redjeb, G. Fournier, and A. Ben Hassen. 1989. Epidemiology of extended-spectrum -lactamases. Infection 17: 347354. 21. Philippon, A., R. Labia, and G. Jacoby. 1989. Extended-spectrum -lactamases. Antimicrob. Agents Chemother. 33: 11311136. 22. Quinn, J. P., D. Miyashiro, D. Sahm, R. Flamm, and K. Bush. 1989. Novel plasmid-mediated -lactamase TEM-10 ; conferring selective resistance to ceftazidime and aztreonam in clinical isolates of Klebsiella pneumoniae. Antimicrob. Agents Chemother. 33: 14511456. 23. Rice, L. B., J. D. C. Yao, K. Klimm, G. M. Eliopoulos, and R. C. Moellering, Jr. 1991. Efficacy of different -lactams against an extended-spectrum -lactamase-producing Klebsiella pneumoniae strain in the rat intra-abdominal abscess model. Antimicrob. Agents Chemother. 35: 12431244. 24. Rice, L. B., S. H. Willey, G. A. Papanicolaou, A. A. Medeiros, G. M. Eliopoulos, R. C. Moellering, Jr., and G. A. Jacoby. 1990. Outbreak of ceftazidime resistance caused by extended-spectrum -lactamases at a Massachusetts chronic-care facility. Antimicrob. Agents Chemother. 34: 21932199. 25. Sanders, C. C., and W. E. Sanders, Jr. 1992. -Lactam resistance in gramnegative bacteria: global trends and clinical impact. Clin. Infect. Dis. 15: 824 839. Sirot, D. L., F. W. Goldstein, C. J. Soussy, A. L. Courtieu, M. O. Husson, J. Lemozy, M. Meyran, C. Morel, R. Perez, C. Quentin-Noury, M. E. Reverdy, J. M. Scheftel, M. Rosembaum, and Y. Rezvani. 1992. Resistance to cefotaxime and seven other -lactams in members of the family Enterobacteriaceae: a 3-year study in France. Antimicrob. Agents Chemother. 36: 1677 1681. Smith, C. E., B. S. Tillman, A. W. Howell, R. N. Longfield, and J. H. Jorgensen. 1990. Failure of ceftazidime-amikacin therapy for bacteremia and meningitis due to Klebsiella pneumoniae producing an extended-spectrum -lactamase. Antimicrob. Agents Chemother. 34: 12901293. 28. Spencer, R. C., P. F. Wheat, T. G. Winstanley, D. M. Cox, and S. J. Plested. 1987. Novel -lactamase in a clinical isolate of Klebsiella pneumoniae conferring unusual resistance to -lactam antibiotics. J. Antimicrob. Chemother. 20: 919927. 29. Thomson, K. S. 1995. Improved detection of extended-spectrum -lactamases ESBLs ; from disk diffusion susceptibility tests, abstr. C-87, p. 16. In Abstracts of the 95th General Meeting of the American Society for Microbiology 1995. American Society for Microbiology, Washington, D.C. 30. Thomson, K. S., and C. C. Sanders. 1992. Detection of extended-spectrum -lactamases in members of the family Enterobacteriaceae: comparison of the double-disk and three-dimensional tests. Antimicrob. Agents Chemother. 36: 18771882. 31. Thornsberry, C. 1995. Trends in antimicrobial resistance among today's bacterial pathogens. Pharmacotherapy 15 1 Pt. 2 ; : 3S8S and requip. To the safety of medicines for treating drug side effects or complications of aging.

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To determine whether the formation of the low mobility RXR-dependent NRRE-1-protein complex was unique to cardiac-derived nuclear proteins, the EMSA were performed with nuclear protein extracts prepared from adult rat brain or liver. Although two light NRRE-1-protein complexes were observed with protein extracts derived from all three tissues in the absence of exogenously added RXR , in the presence of added RXR the prominent low mobility complex was only observed with extracts derived from heart Fig. 2, lanes 1 and 2 compared with lanes 3 6 ; . Moreover, the RXR-dependent complex did not form with nuclear protein extracts prepared from rat skeletal muscle, NIH 3T3 fibroblasts, or the HIB brown adipocyte line data not shown ; . These data identify a cardiac-enriched RXR interacting protein. The Interaction of RXR CERIP with NRRE-1 Requires a Nuclear Receptor Recognition Site--We have shown previously that NRRE-1 is a complex nuclear receptor response element that contains three potential nuclear receptor-binding sites Fig. 3A, sites 1, 2, and 3 ; . EMSAs were performed with a series of mutant NRRE-1 probes to delineate the binding site requirements for the RXR CERIP-DNA complex complex RC ; . Complex RC did not form with mutant NRRE-1 probes containing a deletion of site 1 ; or deletion of sites 2 and 3 2, 3, Fig. 3B, lanes 2 and 3 ; . To explore further the requirements for sites and methotrexate. Pharmacology Actions: Moclobemide is an antidepressant that affects the monoaminergic cerebral neurotransmitter system by means of a reversible inhibition of monoamine oxidase. There are two types of monoamine oxidases, A & B, which vary in their substrate specificity. Moclobemide is relatively selective for type A monoamine oxidase. At a 300 mg dose, the inhibition of monoamine oxidase A is approximately 80% while that of monamine oxidase B is approximately 20-30%. The inhibition is short-lasting approximately 24 hours ; . The metabolism of dopamine, noradrenaline and serotonin is decreased by this effect, and this leads to increased extracellular concentrations of these neuronal transmitters. As a result of its elevating effect on mood and psychomotor activity, moclobemide relieves symptoms such as dysphoria, exhaustion, lack of drive and poor ability to concentrate. These effects most often appear within the first week of therapy. Although moclobemide has no sedative properties it does increase total sleep time. Moclobemide does not impair alertness or reaction time. Moclobemide appears to be suitable for ambulatory treatment as it is not sedating and does not impair vigilance or ability to react. Moclobemide is well tolerated. Short-term and long-term animal studies indicate low toxicity. Little hepatic or cardiac toxicity has been observed. There is a low incidence of raised liver enzymes.

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GB2 Increases Agonist but Not Antagonist ; Affinity in GB1--Although GB1 binds any known GABAB ligands, agonist affinities are 100 times lower than those measured in native receptors 39 ; . This is due partly to GB2 because its coexpression with GB1 results in a 10-fold increase in agonist affinity 4 ; . This effect of GB2 does not result from the targeting of GB1 to the cell surface and so to a mature glycosylation state because a GB1 mutant able to reach the cell surface alone GB1ASA, in which the endoplasmic reticulum retention signal RSRR is mutated into ASAR ; still displayed low agonist affinity at the cell surface Fig. 1b and Table I ; . A direct association between GB1 and GB2 appears therefore necessary to control agonist affinity in this receptor. Coexpression of GB1 with GB2 1, a chimeric subunit composed of GB2 VFT and GB1 HD, or the replacement of GB1 HD with GB2 HD in chimeric GB1 2 also resulted in increased GABA affinity Fig. 1b and Table I ; . These observations suggest that both GB2 VFT and GB1 HD control agonist affinity in GB1. VFT Module Constructs for GB1 and GB2--To elucidate the mechanism leading both GB2 VFT and GB1 HD to allosterically control agonist affinity in GB1, isolated GB1 and GB2 VFTs were prepared by removing the HD and C-terminal domain of each subunit. These constructs were generated to examine the possible direct interaction between GB1 and GB2 VFTs and the consequence on GABA affinity, regardless the.

Eating only five percent of your calories in the evening hours and eating 65 percent of your calories in the evening hours. It turned out that the ones that ate a large percentage of their calories at night were no more likely to gain weight than the ones that ate during the day." Cameron was surprised by the results, she says, because it was a popular notion and the recommendation of many diets to limit food intake in the evening. "It also surprised us that some animals that really increased their food intake were not gaining weight, while others that increased their food intake just a little bit gained weight, " she says. "The total amount of food intake didn't correlate very well with weight gain." Cameron says the best predictor of weight gain is activity. The monkeys in the study that gained the most weight were the ones that just sat around and strattera. Tain calendar; schedule & track appointments; perform copying, word processing, filing, typing, mailing & other general clerical duties; facilitate implementation of planned events; schedule travel & process reimbursements. QUALIFICATIONS: HS diploma, business curriculum or equivalent; minimum 2 years secretarial administrative assistant experience preferred; type 55 wpm; familiarity with word processing packages; evidence of prior, strong commitment to mission of urban education; skilled in Macintosh, Windows & Microsoft Word; willingness to learn Excel & to work cooperatively with other GSE staff in computing; demonstrated ability to supervise work-study students & Research Assistants who are.
Been on purinethol for 10 month and indinavir. Immobility can cause pain, and pain can cause immobility. If it hurts when a person moves, of course they don't want to move. If they don't move, they are at high risk for contractures and decubitus ulcers, which can cause more pain. External factors. This could be as simple as an undergarment that is fastened too tight or a wheelchair leg rest with a rough edge. Look around the resident for easily corrected things that could cause pain. Also, a room that is too cold or hot, a bright light, or loud noises can all make pain harder to tolerate. Emotional factors. Pain does increase anxiety, and anxiety may increase pain. While treating anxiety helps, it shouldn't be treated only with medicines. A warm bath, soft music, hand-holding, hugs, and just listening all can decrease anxiety. Letting the resident know you believe their pain is real and that you want to help is also extremely important.

For this purpose, the cutoff values that we derived are satisfactory 4 ; . They generally provide a clear distinction of microalbuminuria from normal, because the diagnostic values are far removed from the normal population mean 5 ; . We found that the ACR had less within-subject variation than timed overnight measurements 4 ; . For this reason, coupled with its simplicity, we feel that the use of single samples with or without the conversion to 24-h albumin excretion rates, as advocated by Cundy and Baker, is preferable for large surveys and routine clinical use. JOHN N. HARVEY, MD and aricept.
The following is a list of preferred brand medications. It represents the drug list formulary ; that is at the core of your pharmacy benefit plan. This list does not guarantee coverage. The actual benefit will be determined at the time the claim is received. In addition to using this list, you are encouraged to ask your doctor to prescribe generic medications whenever possible. This list is effective January 1, 2005 through December 31, 2005. This list is subject to change. You can get more information and updates to this list at our website at pbmplus PCE PENTASA PHENABID PHOSPHOLINE IODIDE PLAVIX PRANDIN PRECISION XTRA METER PRECISION XTRA TEST STRIP PRECOSE PRED-G PREMARIN PREMPHASE PREMPRO PREVACID PRO-BANTHINE PROCTOCREAM-HC PROCTOCREAM-HC PROCTOFOAM HC PROGRAF PROMETRIUM PROSCAR PROSTIGMIN PROSTIGMIN PROTONIX PROTOPIC PULMICORT PURINETHOL P P P ROWASA.

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In the past four years, drug counterfeiters also planned other attacks which by the grace of God did not succeed. In one of such instances, six 6 ; armed men invaded my residence in August 2001 but fortunately I was not available. There have been numerous other threats through mails, telephone calls, deposition of fetish objects in my office e.t.c. My family members and NAFDAC staff are also constantly under threat.

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41 specimens of cervical mucus, follicular and sperm fluids, and human milk that were examined in Germany. Chlorfenvinphos levels of 13.66, 1.69, 2.02, and 1.89 g kg were detected in 4 of the 11 samples of cervical mucus. Chlorfenvinphos levels of 0.42 g kg were detected in 1 of the 10 sperm fluid samples and 1 of the 10 human milk samples, respectively. The detection of chlorfenvinphos in the cervical mucus, which showed the highest levels, was unexpected because it was believed to be the most unlikely site for accumulation in the body. It was suggested that a connection exists between the activities of the cervical glands of the endocervix and the appearance of some pesticides in the cervical mucus which might show a new way for accumulation. Accordingly, the data indicate that organophosphorus environmental pollutants, like chlorfenvinphos, can appear in the human reproductive organs, exposing even germ-cells and, thus, present a risk of interference with the process of reproduction Wagner et al. 1990 ; . Since the data were generated from environmental exposure, the combined route of exposure may include inhalation. No studies were located regarding the distribution of chlorfenvinphos after inhalation exposure in animals. 2.3.2.2 Oral Exposure Although chlorfenvinphos is a hydrophilic substance, it has hitherto not been widely found in human tissues because it is not expected to persist in these tissues. As an organophosphorus compound, chlorfenvinphos is not expected to accumulate in the body tissues because of its expected short halflife. However, chlorfenvinphos was found in some of the 41 specimens of cervical mucus, follicular and sperm fluids, and human milk that were examined. Chlorfenvinphos levels of 13.66, 1.69, 2.02, and 1.89 g kg were detected in 4 of the 11 samples of cervical mucus. Chlorfenvinphos levels of 0.42 g kg were detected in 1 of the 10 sperm fluid samples and 1 of the 10 human milk samples, respectively. The detection of chlorfenvinphos in the cervical mucus, which showed the highest levels, was unexpected because it is the most unlikely compartment for accumulation in the body. It was suggested that a connection exists between the gland's activities and the appearance of some pesticides in the cervical mucus, which might show a new way for accumulation. Accordingly, these data indicate that new environmental pollutants, like chlorfenvinphos, can appear in the human organs, exposing even germ-cells and, thus, present a risk of adverse effects on reproduction Wagner et al. 1990 ; . Since the data were generated from environmental exposure, the combined route of exposure possibly include oral. A 29-year-old male was hospitalized with severe respiratory distress and bronchial tree hypersecretion. The patient had ingested about 50 ml of the preparation Enolofos, which contains!

Retrospective "data dredging" without an adequate prospective hypothesis Unclear description of study population and inclusion criteria Unclear or inappropriate study design e.g., casecontrol study with disease prevalence of 50% ; Failure to consider prevalence of variant in sample size calculation Failure to consider effect modifier esp. gene-gene interaction ; or other confounders age, BMI, etc.

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Ogy, Washington, D.C. 2. Burke, J. P., R A. Garibaldi, M. R. Britt, J. A. Jacobsen, M. Conti, and A. W. Ailing. 1981. Prevention of catheter-associated urinary tract infections: efficacy of daily meatal care regimens. Am. J. Med. 70: 655-658. 3. Butler, H. K., and C. M. Kunin. 1968. Evaluation of polymyxin catheter lubricant and impregnated catheters. J. Urol. 100: 560566. 4. Classen, D. C., L. E. Stevens, S. A. Bass, and J. P. Burke. 1990. Lack of efficacy of a silver oxide-coated urinary catheter in the prevention of catheter-associated bacteriuria: a large randomized clinical trial, abstr. 710, p. 204. Program Abstr. 30th Intersci. Conf. Antimicrob. Agents Chemother. 5. Daifuku, R, and W. E. Stamm. 1984. Association of rectal and urethral colonization with urinary tract infection in patients with indwelling catheters. JAMA 252: 2028-2030. 6. Desautels, R. E., C. W. Walter, R. C. Graves, and J. H. Harrison. 1962. Technical advances in the prevention of urinary tract infection. J. Urol. 87: 487-490. 7. Duffy, A. M. ed. ; . 1992. Physicians' desk reference, 46th ed., p. 1854-1855. Medical Economics Co., Montvale, N.J. 8. Garibaldi, R. A., J. P. Burke, M. R. Britt, W. A. Miller, and C. B. Smith. 1980. Meatal colonization and catheter-associated bacteriuria. N. Engl. J. Med. 303: 316-318. 9. Givens, C. D., and R. P. Wenzel. 1980. Catheter-associated urinary tract infections in surgical patients: a controlled study of the excess morbidity and costs. J. Urol. 124: 646-648. 10. Green, M. S., E. Rubinstein, and P. Amit. 1982. Estimating the effects of nosocomial infections on the length of hospitalization. J. Infect. Dis. 145: 667-672. 11. Harvey, S. C. 1975. Antiseptics and disinfectants; fungicides; ectoparasiticides, p. 1002-1003. In L. S. Goodman and A. Gilman ed. ; , The pharmacological basis of therapeutics. Macmillan, New York. 12. HuaUin, C. 1988. Manufacture and clinical employment of an antibiotic silicone-rubber catheter. Eur. Urol. 14: 72-74. 13. Huth, T. S., J. P. Burke, R. A. Larsen, D. C. Classen, and L. E. Stevens. 1992. Randomized trial of meatal care with silver sulfadiazine cream for the prevention of catheter-associated bacteriuria. J. Infect. Dis. 165: 14-18. 14. Johnson, J. R., T. Berggren, and A. J. Conway. 1992. Activity of a nitrofurazone silicone matrix urinary catheter against catheter-associated uropathogens, abstr. 1140, p. 301. Program Abstr. 32nd Intersci. Conf. Antimicrob. Agents Chemother. 15. Johnson, J. R., P. L. Roberts, R. J. Olsen, K. A. Moyer, and W. E. Stamm. 1990. Prevention of catheter-associated urinary tract infection with a silver-oxide urinary catheter: clinical and microbiologic correlates. J. Infect. Dis. 162: 1145-1150. 16. Krieger, J. N., D. L. Kaiser, and R. P. Wenzel. 1983. Urinary tract etiology of bloodstream infection in hospitalized patients. J. Infect. Dis. 148: 57-62. 17. Kunin, C. M., and R. C. McCormacL 1966. Prevention of catheter-induced urinary-tract infections by sterile closed drainage. N. Engl. J. Med. 274: 1155-1160. 18. Lazarus, S. M., J. N. LaGuerre, H. Kay, S. Weinberg, and B. S. Levowitz. 1971. A hydrophilic polymer-coated antimicrobial urethral catheter. J. Biomed. Mater. Res. 5: 129-138. 19. Lennette, E. H., A. Balows, W. J. Hausler, Jr., and H. J. Shadomy ed. ; . 1985. Manual of clinical microbiology, 4th ed. American Society for Microbiology, Washington, D.C. 20. Lundeberg, T. 1986. Prevention of catheter-associated urinarytract infections by use of silver-impregnated catheters. Lancet i: 1031. Letter ; . 21. Monson, T., and C. M. Kunin. 1974. Evaluation of a polymercoated indwelling catheter in prevention of infection. J. Urol. 111: 220-222. 22. National Committee for Clinical Laboratory Standards. 1990. Methods for dilution antimicrobial susceptibility tests for bacteria that grow aerobically, 2nd ed. Approved standard. NCCLS document M7-A2. National Committee for Clinical Laboratory Standards, Villanova, Pa. 23. National Committee for Clinical Laboratory Standards. 1990.

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